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ISSN 2063-5346
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Brief Insight about Intracerebral hemorrhage

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Ahmed Essam Mohamed Harby, Sawsan Abdel aziz yousef, Wafaa Samir Mohamed, Emad Latif Agban
» doi: 10.31838/ecb/2023.12.1.459

Abstract

the World Health Organization (WHO) defined stroke as “rapidly developing clinical signs of focal disturbance of cerebral function, lasting more than 24 hours or leading to death with no apparent cause other than that of vascular origin”. The pathology underlying strokes includes infarctions with its anatomical subdivisions [(Small vessel (lacunar) infarction, total or partial territorial infarction and border-zone infarction)], Intracerebral haemorrhage (ICH) with its anatomical subdivision (lobar, deep/basal ganglia and posterior fossa), Subarachnoid haemorrhage either aneurysmal or secondary to arteriovenous malformation (AVM) and Cerebral venous sinus thrombosis. The majority of strokes result from arterial pathology but a small proportion, less than 1%, results from cerebral venous thrombosis. Intracerebral hemorrhage (ICH) is the second most common type of stroke, after ischemic stroke with high morbidity and mortality. 30-day mortality for ICH is about 35–52 % with one-half of the deaths occuring during the acute phase, especially within the first 2 days. Intracerebral hemorrhage (ICH) is caused by bleeding, primarily into brain tissue parenchyma. Underlying pathologies are classified into arterial (small and large-vessel disease), venous disease, vascular malformation, hemostatic disorders and ICH due to other disorders.

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