Preeclampsia is a clinical condition whose aetiology is described to be multifactorial and the cellular mechanism of which has not been understood completely. Recent studies hugely concentrate on microRNAs due to their regulatory effect on the target gene at the epigenetic level. Since Epithelial-Mesenchymal Transition (EMT) is a common mechanism observed both in cancer cell lines and embryological development and miR200 is a regulator of EMT, a large body of studies has been carried out in this regard. Previous studies have shown the regulatory effect of miR200 on ZEB1/ZEB2 mRNA in EMT. Since the trophoblast invasion is found to be a deficit in Preeclampsia, the expression level of miR200 requires detailed studies. The present study shows that miR210 expression is significantly upregulated in preeclamptic placental tissue in comparison with age-matched normotensive controls in the rt-PCR study. Further studies are needed to explore whether the therapeutic regulation of miR200 reverses the EMT suppression imposed by miRNA200.